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reference strain atcc baa1605  (ATCC)


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    Structured Review

    ATCC reference strain atcc baa1605
    Reference Strain Atcc Baa1605, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 544 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/reference+strain+atcc+baa1605/Acinetobacter+baumannii+Bouvet+and+Grimont/pm41599086-166-23-25
    Average 96 stars, based on 544 article reviews
    reference strain atcc baa1605 - by Bioz Stars, 2026-09
    96/100 stars

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    Related Articles

    Functional Assay:

    Article Title: Inside Enemy Lines: Adhesion, Invasion, and Intracellular Persistence of Acinetobacter baumannii in the Respiratory Epithelium
    Article Snippet: In strain ATCC 17978, genes associated with adhesion to A549 human alveolar epithelial cells and involved in iron uptake, including feoA (encoding the ferrous iron transport system component FeoA), bnfL (encoding an N-acetyltransferase, baumannoferrin biosynthetic protein), basB (encoding an acinetobactin biosynthesis protein), were also involved in in vivo virulence during pneumonia infection, along with mtnN (encoding an MTA/SAH nucleosidase), yfgC (renamed bepA , encoding a putative periplasmic metalloprotease), hisF (encoding the imidazole glycerol phosphate synthase cyclase subunit), and oatA (encoding an acyltransferase) [ ]. .. Accordingly, recent transcriptomic and functional analyses performed in vivo in the G. mellonella infection model with the community strain Ab-C98 and the clinical reference strain ATCC BAA1605 showed a strong upregulation of genes belonging to the acinetobactin, baumannoferrin, and feo iron clusters [ ]. .. Further functional validation by gene knockout showed that the deletion of the siderophore biosynthesis genes ( basC , bfnD , and basF ) significantly increased host survival by confirming that these determinants contribute to A. baumannii virulence.

    Article Title: Inside Enemy Lines: Adhesion, Invasion, and Intracellular Persistence of Acinetobacter baumannii in the Respiratory Epithelium.
    Article Snippet: In strain ATCC 17978, genes associated with adhesion to A549 human alveolar epithelial cells and involved in iron uptake, including feoA (encoding the ferrous iron transport system component FeoA), bnfL (encoding an N-acetyltransferase, baumannoferrin biosynthetic protein), basB (encoding an acinetobactin biosynthesis protein), were also involved in in vivo virulence during pneumonia infection, along with mtnN (encoding an MTA/SAH nucleosidase), yfgC (renamed bepA, encoding a putative periplasmic metalloprotease), hisF (encoding the imidazole glycerol phosphate synthase cyclase subunit), and oatA (encoding an acyltransferase) [57]. .. Accordingly, recent transcriptomic and functional analyses performed in vivo in the G. mellonella infection model with the community strain Ab-C98 and the clinical reference strain ATCC BAA1605 showed a strong upregulation of genes belonging to the acinetobactin, baumannoferrin, and feo iron clusters [86]. .. Further functional validation by gene knockout showed that the deletion of the siderophore biosynthesis genes (basC, bfnD, and basF) significantly increased host survival by confirming that these determinants contribute to A. baumannii virulence.

    In Vivo:

    Article Title: Inside Enemy Lines: Adhesion, Invasion, and Intracellular Persistence of Acinetobacter baumannii in the Respiratory Epithelium
    Article Snippet: In strain ATCC 17978, genes associated with adhesion to A549 human alveolar epithelial cells and involved in iron uptake, including feoA (encoding the ferrous iron transport system component FeoA), bnfL (encoding an N-acetyltransferase, baumannoferrin biosynthetic protein), basB (encoding an acinetobactin biosynthesis protein), were also involved in in vivo virulence during pneumonia infection, along with mtnN (encoding an MTA/SAH nucleosidase), yfgC (renamed bepA , encoding a putative periplasmic metalloprotease), hisF (encoding the imidazole glycerol phosphate synthase cyclase subunit), and oatA (encoding an acyltransferase) [ ]. .. Accordingly, recent transcriptomic and functional analyses performed in vivo in the G. mellonella infection model with the community strain Ab-C98 and the clinical reference strain ATCC BAA1605 showed a strong upregulation of genes belonging to the acinetobactin, baumannoferrin, and feo iron clusters [ ]. .. Further functional validation by gene knockout showed that the deletion of the siderophore biosynthesis genes ( basC , bfnD , and basF ) significantly increased host survival by confirming that these determinants contribute to A. baumannii virulence.

    Article Title: Inside Enemy Lines: Adhesion, Invasion, and Intracellular Persistence of Acinetobacter baumannii in the Respiratory Epithelium.
    Article Snippet: In strain ATCC 17978, genes associated with adhesion to A549 human alveolar epithelial cells and involved in iron uptake, including feoA (encoding the ferrous iron transport system component FeoA), bnfL (encoding an N-acetyltransferase, baumannoferrin biosynthetic protein), basB (encoding an acinetobactin biosynthesis protein), were also involved in in vivo virulence during pneumonia infection, along with mtnN (encoding an MTA/SAH nucleosidase), yfgC (renamed bepA, encoding a putative periplasmic metalloprotease), hisF (encoding the imidazole glycerol phosphate synthase cyclase subunit), and oatA (encoding an acyltransferase) [57]. .. Accordingly, recent transcriptomic and functional analyses performed in vivo in the G. mellonella infection model with the community strain Ab-C98 and the clinical reference strain ATCC BAA1605 showed a strong upregulation of genes belonging to the acinetobactin, baumannoferrin, and feo iron clusters [86]. .. Further functional validation by gene knockout showed that the deletion of the siderophore biosynthesis genes (basC, bfnD, and basF) significantly increased host survival by confirming that these determinants contribute to A. baumannii virulence.

    Infection:

    Article Title: Inside Enemy Lines: Adhesion, Invasion, and Intracellular Persistence of Acinetobacter baumannii in the Respiratory Epithelium
    Article Snippet: In strain ATCC 17978, genes associated with adhesion to A549 human alveolar epithelial cells and involved in iron uptake, including feoA (encoding the ferrous iron transport system component FeoA), bnfL (encoding an N-acetyltransferase, baumannoferrin biosynthetic protein), basB (encoding an acinetobactin biosynthesis protein), were also involved in in vivo virulence during pneumonia infection, along with mtnN (encoding an MTA/SAH nucleosidase), yfgC (renamed bepA , encoding a putative periplasmic metalloprotease), hisF (encoding the imidazole glycerol phosphate synthase cyclase subunit), and oatA (encoding an acyltransferase) [ ]. .. Accordingly, recent transcriptomic and functional analyses performed in vivo in the G. mellonella infection model with the community strain Ab-C98 and the clinical reference strain ATCC BAA1605 showed a strong upregulation of genes belonging to the acinetobactin, baumannoferrin, and feo iron clusters [ ]. .. Further functional validation by gene knockout showed that the deletion of the siderophore biosynthesis genes ( basC , bfnD , and basF ) significantly increased host survival by confirming that these determinants contribute to A. baumannii virulence.

    Article Title: Inside Enemy Lines: Adhesion, Invasion, and Intracellular Persistence of Acinetobacter baumannii in the Respiratory Epithelium.
    Article Snippet: In strain ATCC 17978, genes associated with adhesion to A549 human alveolar epithelial cells and involved in iron uptake, including feoA (encoding the ferrous iron transport system component FeoA), bnfL (encoding an N-acetyltransferase, baumannoferrin biosynthetic protein), basB (encoding an acinetobactin biosynthesis protein), were also involved in in vivo virulence during pneumonia infection, along with mtnN (encoding an MTA/SAH nucleosidase), yfgC (renamed bepA, encoding a putative periplasmic metalloprotease), hisF (encoding the imidazole glycerol phosphate synthase cyclase subunit), and oatA (encoding an acyltransferase) [57]. .. Accordingly, recent transcriptomic and functional analyses performed in vivo in the G. mellonella infection model with the community strain Ab-C98 and the clinical reference strain ATCC BAA1605 showed a strong upregulation of genes belonging to the acinetobactin, baumannoferrin, and feo iron clusters [86]. .. Further functional validation by gene knockout showed that the deletion of the siderophore biosynthesis genes (basC, bfnD, and basF) significantly increased host survival by confirming that these determinants contribute to A. baumannii virulence.

    Article Title: Transcriptomic insights into the virulence of Acinetobacter baumannii during infection-role of iron uptake and siderophore production genes.
    Article Snippet: .. To compare the killing of Ab-C98 with hospital strain A. baumannii, in this study, we performed a G. mellonella killing assay and bacterial burden assay for the reference strain ATCC BAA1605 (Fig. 1), a clinical strain exhibited higher virulence than other ATCC strains (ATCC 17978 and ATCC 19606) in the mouse infection model [47]. .. Interestingly, the same infection dose of 107 CFU of ATCC BAA1605 (109 CFU mL 1) 5FEBS Letters (2024) a 2024 Federation of European Biochemical Societies. nloaded from https://febs.onlinelibrary.w iley.com /doi/10.1002/1873-3468.15061 by C A PE S, W iley O nline L ibrary on [04/12/2024].



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